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Toxicant-Receptor Interactions: Modulations of signal transduction and gene expression (Target Organ Toxicology Series)
 
 
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Toxicant-Receptor Interactions: Modulations of signal transduction and gene expression (Target Organ Toxicology Series) [Hardcover]

Michael Denison (Editor), William Helferich (Editor)

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Book Description

1560326336 978-1560326335 August 10, 1998 1
Many of the toxic effects elicited by xenobiotics can be explained at the molecular level by their interaction with receptors or by disruption or interference with receptor-mediated signal transduction pathways. This volume describes molecular approaches and reviews of current research. It provides reviews of numerous research areas which are directly related to the analysis of the molecular mechanism by which xenobiotics modulate signal transduction and gene expression.

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Inside This Book (learn more)
First Sentence:
Halogenated aromatic hydrocarbons (HAHs), such as polychlorinated dibenzo-pdioxins (PCDDs), biphenyls (PCBs). Read the first page
Key Phrases - Statistically Improbable Phrases (SIPs): (learn more)
various detoxifying enzyme genes, barbie box elements, positive regulator proteins, ryanodme receptor, positive regulatory proteins, mouse hepatoma cells, scarce component, hepatic plasma membrane, receptor transformation, electronic subtraction, antiestrogenic activity, pleiotropic response, other barbiturates, expression assays, peroxisome proliferation, peroxisomal proliferation, estrogen agonists, estrogenic compounds, occurring fatty acids, dioxin receptor, lipid homeostasis, hydrocarbon receptor, human breast cancer cells, antiestrogenic activities, antiestrogenic effects
Key Phrases - Capitalized Phrases (CAPs): (learn more)
Biol Chem, Proc Natl Acad Sci, Toxicol Appl Pharmacol, New York, Cancer Res, Environ Health Perspect, Biochem Pharmacol, Mol Cell Biol, Mol Pharmacol, Proc Nail Acad Sci, Blot Chem, Nucleic Acids Res, Mot Pharmacol, Vanden Heuvel, Arch Biochem Biophys, Mot Cell Blot, Biochem Biophys Res Commun, Helferich Copyright, Mot Endocrinol, Toxicol Lett, Mol Cell Biochem, Plenum Press, Biochem Biophys Acta, Biochim Biophys Acta, Biol Client
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