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Peak K2
 
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Peak K2

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Product Features

  • Vitamin K is an essential nutrient known for its role in blood clotting. Plants make one form of vitamin K (phylloquinone, or vitamin K1) for their use. But your body doesnt use all of the K1 in your diet as is. Instead, the body converts some of this plant form of the vitamin into a different vitamin K molecule: Menatetrenone, or MK-4, a form of vitamin K2.. For some purposes (blood clotting), K1 works fine; but Menatetrenone has unique effects on bone health not shared by phylloquinone
  • Fracture victims levels of Menatetrenone are more depressed than are their levels of phylloquinone
  • Menatetrenone is able to reduce the creation of osteoclasts (cells involved in the teardown of bone tissue) out of early cell types but again, phylloquinone has no such power.
  • Menatetrenone strengthens the bone-building legions of the osteoblasts (cells involved in the manufacture of new bone), mildly increasing both their numbers and their activity.
  • Areas where more K2 is consumed in the diet have lower fracture rates.


Product Description

Product Description
By AOR-Canada. Over the course of the last decade, at least sixteen clinical trials have been performed using Menatetrenone, and every single one has found that K2 supplements protect bone health. Menatetrenone not only slows, halts, or even reverses loss of bone mass: it dramatically reduces your risk of suffering a fracture. In one trial, Menatetrenone was put to the test in a direct comparison against the bisphosphonate drug etidronate (Didrocal®). Menatetrenone preserved bone mass, and also slashed fracture risk by roughly two thirds over the course of two years. In another trial, osteoporotic women taking Menatetrenone supplements sustained nearly no bone loss over two years, while cutting fracture risk by 64% as compared with non-supplementing women. The ability of bones to withstand fractures is not just determined by the quantity of bone (as measured by Bone Mineral Density (BMD)), but also by the quality of bone bone microarchitecture, including trabecular connectivity. Evidence suggests that Menatetrenones most important effects are on bone quality, not quantity. Clinical trials have found that Menatetrenone provides as much protection against fracture as drugs that have much more powerful effects on BMD. Clearly, Menatetrenones bone-protective effects extend to aspects of bone health beyond the BMD numbers.Menatetrenone provides powerful protection against the loss of trabecular connectivity in laboratory animal models of menopausal osteoporosis

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